Prasad was right. And Cheng - particularly the first... pubmed.ncbi.nlm.nih.gov… and pubmed.ncbi.nlm.nih.gov… and pubmed.ncbi.nlm.nih.gov… as much as said something similar without saying the same thing.

I note though that the industry was anxious to refute that: pubmed.ncbi.nlm.nih.gov…

Which is ridiculous, because cov-2 absolutely has the potential to be a superantigen in certain people especially in the existence of pre-existing prolonged stress which is crucial for spike unfolding and binding efficiently pubmed.ncbi.nlm.nih.gov… .

Quote" In other words, the formation of disulfide bonds, prevalent during oxidative stress, creates a conformation ready to bind to the receptor. Taken together, the present study demonstrates the role of pre-existing oxidative stress in elevating the binding affinity of the spike protein for the human receptor, offering future clues for alternate therapeutic possibilities."

And how would you create that? Create massive year long psycho social stress of course. This article doesn't say that directly but it does say that psycho social stress creates ROS, and ROS is what is needed to increase the binding affinity.

pubmed.ncbi.nlm.nih.gov…

I'm sure there are a whole lot more needles in the haystack we could find as time goes on.