The coronavirus can't benefit from that logic. There is no mechanistic basis.

Herpes viruses dedicate the majority of their genome to sophisticated immune evasion timers that turn off after infiltrating various niches within the body, and even appear to enhance immune function against other viruses (to me; most of the research interprets this behavior negatively in spite of the fact that we're all walking around with these viruses all our lives and are perfectly fine). It is these sophisticated timer genes which get distorted by passage through "leaky" immunity, leading to unvaccinated host death.

There's nothing like that in the coronavirus phenotype.